signaling cascade Search Results


ezyamp signal amplification cascade  (SpeeDx Pty Ltd)
90
SpeeDx Pty Ltd ezyamp signal amplification cascade
Ezyamp Signal Amplification Cascade, supplied by SpeeDx Pty Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ezyamp signal amplification cascade - by Bioz Stars, 2026-10
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metacore pathway map for development leptin signaling via jak/stat and mitogen-activated protein kinase (mapk) cascades  (GeneGo Inc)
90
GeneGo Inc metacore pathway map for development leptin signaling via jak/stat and mitogen-activated protein kinase (mapk) cascades
Metacore Pathway Map For Development Leptin Signaling Via Jak/Stat And Mitogen Activated Protein Kinase (Mapk) Cascades, supplied by GeneGo Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
metacore pathway map for development leptin signaling via jak/stat and mitogen-activated protein kinase (mapk) cascades - by Bioz Stars, 2026-10
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intestinal pmf signaling cascade  (TransTissue Technologies GmbH)
90
TransTissue Technologies GmbH intestinal pmf signaling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Intestinal Pmf Signaling Cascade, supplied by TransTissue Technologies GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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intestinal pmf signaling cascade - by Bioz Stars, 2026-10
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signalling cascade  (Millar Inc)
90
Millar Inc signalling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Signalling Cascade, supplied by Millar Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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signalling cascade - by Bioz Stars, 2026-10
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a (pka) signaling cascade  (ProteinKinase)
90
ProteinKinase a (pka) signaling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
A (Pka) Signaling Cascade, supplied by ProteinKinase, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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sbml file containing the thermodynamically feasible egf/erk signaling cascade model  (Biomodels LLC)
90
Biomodels LLC sbml file containing the thermodynamically feasible egf/erk signaling cascade model
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Sbml File Containing The Thermodynamically Feasible Egf/Erk Signaling Cascade Model, supplied by Biomodels LLC, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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inhibitors of intracellular signaling cascades  (Biomol GmbH)
90
Biomol GmbH inhibitors of intracellular signaling cascades
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Inhibitors Of Intracellular Signaling Cascades, supplied by Biomol GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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signaling cascade  (Agouron Inc)
90
Agouron Inc signaling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Signaling Cascade, supplied by Agouron Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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signaling cascade - by Bioz Stars, 2026-10
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jak^stat signaling cascade  (Federation of European Neuroscience Societies)
90
Federation of European Neuroscience Societies jak^stat signaling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Jak^Stat Signaling Cascade, supplied by Federation of European Neuroscience Societies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/signaling+cascade/jak+stat+signaling+cascade/pm11163768-6-11-43
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flowcellecttm pi3k-mtor signalling cascade kit #fccs025210  (Merck KGaA)
90
Merck KGaA flowcellecttm pi3k-mtor signalling cascade kit #fccs025210
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Flowcellecttm Pi3k Mtor Signalling Cascade Kit #Fccs025210, supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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a signaling cascade  (ProteinKinase)
90
ProteinKinase a signaling cascade
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
A Signaling Cascade, supplied by ProteinKinase, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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camp/pka ca2þ/pkc signaling cascades  (Informa UK Limited)
90
Informa UK Limited camp/pka ca2þ/pkc signaling cascades
( A ) Schematic of speed increase of the animal on food initiated by <t>PMF</t> dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of <t>mutant</t> <t>AMPK</t> worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.
Camp/Pka Ca2þ/Pkc Signaling Cascades, supplied by Informa UK Limited, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


( A ) Schematic of speed increase of the animal on food initiated by PMF dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of mutant AMPK worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.

Journal: Science Advances

Article Title: Mitochondrial energy state controls AMPK-mediated foraging behavior in C. elegans

doi: 10.1126/sciadv.adm8815

Figure Lengend Snippet: ( A ) Schematic of speed increase of the animal on food initiated by PMF dissipation. ( B ) Frequencies of body bends on food worms treated with FCCP. Two-tailed unpaired t test was performed. Data are means ± SD, n = 68 animals collected in 3 days. ( C ) Change of body bends of mutant AMPK worms expressing ubiquitous mtOFF with AMPK rescue in the indicated neurons. The graph shows the optogenetic active condition (+ATR +light) normalized to the mean of the dark condition (+ATR, −light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are displayed in figs. S7 and S8. Promoters drive expression as follows: UPN , unc-119 , panneuronal; ttx-3 , AIY interneurons; flp-15 , I2 interneurons; npr-9 , AIB interneurons; sra-6 , ASH neurons; rig-3 , AVA interneurons; gcy-28.d , AIA interneurons; dat-1 , dopaminergic neurons; and tph -1, serotonergic neurons. One-way analysis of variance (ANOVA) with Tukey’s test was applied. Data are means ± SD, n = 24 animals collected in 3 days. ( D and E ) Frequency of reversals and omega turns in mutant AMPK worms ubiquitously expressing mtOFF with rescued AMPK in serotonergic (D) or dopaminergic (E) neurons off-food. Kruskal-Wallis test with Dunn’s test was performed. Data are means ± SD, n = 23 to 29 animals collected in 4 days. ( F and G ) Change in behavior in response to PMF dissipation. All data collected in four experimental days were used, and the animals that did not perform any omega turn/reversal were excluded. The optogenetic active condition (+ATR, +light) of each worm was normalized to the mean of the corresponding control condition (−ATR, +light) using the formula: [(+A+L)–mean(−A+L)]/mean(−A+L). The raw data are provided in , . Kruskal-Wallis test with Dunn’s test was applied.

Article Snippet: These findings overall indicate that the intestinal PMF signaling cascade requires neuronal AMPK to facilitate foraging behavior in a transtissue manner.

Techniques: Two Tailed Test, Mutagenesis, Expressing, Control